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Regulation of procaspase-2 by glucocorticoid modulatory element-binding protein 1 through the interaction with caspase recruitment domain.

Abstract：

:Caspases are the primary executioners of apoptosis. Although procaspases believe to exist as inactive forms in cells, the detailed regulatory system remains unclear. Here we show that glucocorticoid modulatory element-binding protein 1 (GMEB1) is capable of binding to the prodomain of caspase-2. We found that this molecule inhibits the autoproteolytic activation of procaspase-2 by oligomerization on a chemical compound-dependent system. These findings indicated that GMEB1 might be an endogenous inhibitory protein that selectively interacts with prodomain of caspase-2 to disrupt the autoactivation.

journal_name

Biochem Biophys Res Commun

journal_title

Biochemical and biophysical research communications

authors

Tsuruma K,Nakagawa T,Shirakura H,Hayashi N,Uehara T,Nomura Y

doi

10.1016/j.bbrc.2004.10.145

subject

Has Abstract

pub_date

2004-12-24 00:00:00

pages

1246-51

issue

4

eissn

0006-291X

issn

1090-2104

pii

S0006-291X(04)02488-X

journal_volume

325

pub_type

杂志文章

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