Pathophysiology of supraventricular tachycardia.

Abstract:

:Supraventricular tachycardia is due to altered automaticity, triggered activity or most commonly, reentry. Atrial flutter is most frequently due to reentry with counterclockwise activation in the right atrium with the left atrium acting as a bystander. The small wavefronts demonstrated during atrial fibrillation are less random than was previously thought. In the study of atrioventricular (AV) nodal reentrant tachycardia, recent evidence suggests that multiple pathways involving intranodal and perinodal tissue are probably involved. In the Wolff-Parkinson-White (WPW) syndrome, accessory pathways near the AV node have been demonstrated with decremental properties, suggesting conduction through accessory AV nodal tissue. The mechanism for atrial fibrillation in the WPW syndrome is poorly understood, but there is evidence that accessory pathways are branched and may themselves support micro-reentry. In Mahaim nodoventricular pathways, the atrium is not essential for reentry, unlike atriofascicular reentrant circuits. Atriofascicular pathways consist of AV node-like tissue and are usually located in the right atrial free wall.

journal_name

Eur Heart J

journal_title

European heart journal

authors

Shakespeare CF,Anderson M,Camm AJ

doi

10.1093/eurheartj/14.suppl_e.2

subject

Has Abstract

pub_date

1993-09-01 00:00:00

pages

2-8

eissn

0195-668X

issn

1522-9645

journal_volume

14 Suppl E

pub_type

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