Abstract：
:Viruses represent some of the deadliest pathogens known to science. Recently they have been reported to have mutualistic interactions with their hosts, providing them direct or indirect benefits. The mutualism and symbiogenesis of such viruses with lower eukaryotic partners such as fungi, yeast, and insects have been reported but the full mechanism of interaction often remains an enigma. In many instances, these viral interactions provide resistance against several biotic and abiotic stresses, which could be the prime reason for the ecological success and positive selection of the hosts. These viruses modulate host metabolism and behavior, so both can obtain maximum benefits from the environment. They bring about micro- and macro-level changes in the hosts, benefiting their adaptation, reproduction, development, and survival. These virus-host interactions can be bilateral or tripartite with a variety of interacting partners. Exploration of these interactions can shed light on one of the well-coordinated biological phenomena of co-evolution and can be highly utilized for various applications in agriculture, fermentation and the pharmaceutical industries.
journal_name
Arch Viroljournal_title
Archives of virologyauthors
Jagdale SS,Joshi RSdoi
10.1007/s00705-017-3686-5subject
Has Abstractpub_date
2018-04-01 00:00:00pages
821-830issue
4eissn
0304-8608issn
1432-8798pii
10.1007/s00705-017-3686-5journal_volume
163pub_type
杂志文章,评审abstract：:Molecular variability was assessed for 18 isolates of cowpea mild mottle virus (CPMMV, genus Carlavirus, family Betaflexiviridae) found infecting soybean in various Brazilian states (Bahia, Goiás, Maranhão, Mato Grosso, Minas Gerais, Pará) in 2001 and 2010. A variety of symptoms was expressed in soybean cv. CD206, ran...
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doi：10.1007/s00705-013-1879-0
更新日期：2014-04-01 00:00:00
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doi：10.1007/s00705-015-2335-0
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s00705-015-2682-x
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更新日期：2017-04-01 00:00:00
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pub_type： 杂志文章
doi：10.1007/s00705-016-2991-8
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doi：10.1007/BF01311694
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s007050050009
更新日期：2000-01-01 00:00:00
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pub_type： 杂志文章
doi：10.1007/BF01316678
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pub_type： 杂志文章
doi：10.1007/s007050170038
更新日期：2001-10-01 00:00:00
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pub_type： 杂志文章
doi：10.1007/s00705-015-2701-y
更新日期：2016-03-01 00:00:00
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s00705-016-3122-2
更新日期：2017-02-01 00:00:00
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s00705-006-0931-8
更新日期：2007-01-01 00:00:00
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s007050050510
更新日期：1999-01-01 00:00:00
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pub_type： 杂志文章
doi：10.1007/BF01316895
更新日期：1993-01-01 00:00:00
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pub_type： 杂志文章
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pub_type： 杂志文章
doi：10.1007/BF01314027
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s00705-014-2241-x
更新日期：2015-01-01 00:00:00
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journal_title：Archives of virology
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journal_title：Archives of virology
pub_type： 杂志文章
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s00705-013-1810-8
更新日期：2014-02-01 00:00:00
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/BF01309681
更新日期：1992-01-01 00:00:00
abstract：:3Y1 rat fibroblasts transformed by adenovirus type 12 or by its E1A gene formed syncytia when cocultivated with the Friend as well as other ecotropic murine leukemia-virus-producing cells. In contrast, the parental 3Y1 cells and those transformed by other tumor viruses or by a chemical carcinogen exhibited no syncytiu...
journal_title：Archives of virology
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doi：10.1007/BF01310628
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更新日期：2008-01-01 00:00:00
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/s00705-006-0910-0
更新日期：2007-01-01 00:00:00
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pub_type： 杂志文章
doi：10.1007/s007050050361
更新日期：1998-01-01 00:00:00
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journal_title：Archives of virology
pub_type： 杂志文章
doi：10.1007/BF01309730
更新日期：1995-01-01 00:00:00
