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Arachidonic acid metabolites regulate interleukin-1 production.

Abstract：

:We have investigated the role of arachidonic acid metabolites in the regulation of interleukin-1 production by murine peritoneal macrophages. Indomethacin a potent inhibitor of prostaglandin synthesis caused a dose-dependent augmentation of lipopolysaccharide induced interleukin production (up to 7-fold at 5 microM). In contrast, lipoxygenase inhibitors, nordihydroguarietic acid and nafazatrom had no effect at doses that did not significantly decrease prostaglandin synthesis. Added to lipopolysaccharide stimulated cultures, PGE2 was also augmented by indomethacin but unlike lipopolysaccharide treated cultures was suppressed by nordihydroguarietic acid. These data suggest that arachidonate metabolites may be potent autoregulators of macrophage interleukin-1 production.

journal_name

Biochem Biophys Res Commun

journal_title

Biochemical and biophysical research communications

authors

Kunkel SL,Chensue SW

doi

10.1016/0006-291x(85)90130-5

subject

Has Abstract

pub_date

1985-04-30 00:00:00

pages

892-7

issue

2

eissn

0006-291X

issn

1090-2104

pii

0006-291X(85)90130-5

journal_volume

128

pub_type

杂志文章

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