Reversible hypomagnesaemia-induced subacute cerebellar syndrome.

Abstract:

:Magnesium is the second most abundant intracellular cation and is a fundamental cofactor in a multitude of cellular enzymatic reactions. Magnesium deficiency causes diverse clinical features predominantly due to cardio- and neurotoxicity. We describe a case of severe hypomagnesaemia associated with intermittent downbeat nystagmus, cerebellar ataxia, generalised convulsions and a supraventricular tachycardia. On MRI imaging, a transient lesion of the cerebellar nodulus was observed, which has not, to our knowledge, been previously described in isolated hypomagnesaemia.

journal_name

Biol Trace Elem Res

authors

Sedehizadeh S,Keogh M,Wills AJ

doi

10.1007/s12011-010-8757-3

subject

Has Abstract

pub_date

2011-08-01 00:00:00

pages

127-9

issue

2

eissn

0163-4984

issn

1559-0720

journal_volume

142

pub_type

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