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Increased cell surface exposure of fucose residues is a late event in apoptosis.

Abstract：

:Apoptosis is a well defined physiological process characterised by many specific features including DNA fragmentation and protease activation. Cell membrane-associated changes such as the altered exposure of phosphatidylserine and glycosylation patterns have also been described. We investigate here the change in exposure of surface alpha-L-fucose residues during thymocyte and P815 cell apoptosis. We show that apoptosis in these cells induced by dexamethasone, gliotoxin or thapsigargin was associated with an increase in the exposure of terminal fucose residues. Furthermore, this increase in fucose exposure occurred late in the apoptotic process. The observation of increased fucose exposure in two different cell types by three different apoptosis-inducing agents suggests it may be part of the normal apoptotic process.

journal_name

Biochem Biophys Res Commun

journal_title

Biochemical and biophysical research communications

authors

Russell L,Waring P,Beaver JP

doi

10.1006/bbrc.1998.9337

subject

Has Abstract

pub_date

1998-09-18 00:00:00

pages

449-53

issue

2

eissn

0006-291X

issn

1090-2104

pii

S0006-291X(98)99337-8

journal_volume

250

pub_type

杂志文章

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