Impairment of cell division of Trypanosoma cruzi epimastigotes.

Abstract:

:The mechanisms that facilitate the adaptation of Trypanosoma cruzi to two distinct hosts, insect and vertebrate, are poorly understood, in part due to the limited ability to perform gene disruption studies by homologous recombination. This report describes a developmentally-defective phenotype that resulted from integration of a drug marker adjacent to the GAPDH gene in T. cruzi.

journal_name

Mem Inst Oswaldo Cruz

authors

Zacks MA

doi

10.1590/s0074-02762007000100019

subject

Has Abstract

pub_date

2007-02-01 00:00:00

pages

111-5

issue

1

eissn

0074-0276

issn

1678-8060

pii

S0074-02762007000100019

journal_volume

102

pub_type

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