Abstract:
:Sepsis was a systemic response to a local infection. Apoptosis was observed in the experimental sepsis. In this study, cecal ligation and puncture (CLP)-induced sepsis was established in rats. We found that sepsis decreased thyroid hormone levels, including triiodothyronine (T3), thyroxine (T4), free T3 (fT3), and free T4 (fT4). Besides, we detected the increasing expression level of Caspase-3 and increasing ratio of TUNEL positive cells in the thyroid after sepsis. Furthermore, a series of pathological ultrastructural changes were observed in thyroid follicular epithelial cells by CLP-induced sepsis. This study established a sepsis animal model and provided the cellular and molecular basis for decoding the pathological mechanism in thyroid with the occurrence of sepsis.
journal_name
Tissue Celljournal_title
Tissue & cellauthors
Lin X,Shi S,Shi Sdoi
10.1016/j.tice.2016.07.001subject
Has Abstractpub_date
2016-10-01 00:00:00pages
511-5issue
5eissn
0040-8166issn
1532-3072pii
S0040-8166(16)30145-8journal_volume
48pub_type
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