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Obstructive sleep and atrial fibrillation: Pathophysiological mechanisms and therapeutic implications.

Abstract：

:Atrial fibrillation (AF) is the commonest arrhythmia in clinical practice and is associated with increased cardiovascular morbidity and mortality. Obstructive sleep apnea (OSA), a common breathing disorder, is an independent risk factor for AF. Several pathophysiological mechanisms, including apnea-induced hypoxia, intrathoracic pressure shifts, sympathovagal imbalance, atrial remodeling, oxidative stress, inflammation and neurohumoral activation have been implicated in the occurrence of AF in OSA patients. In addition, OSA has been shown to reduce success rates of antiarrhythmic drugs, electrical cardioversion and catheter ablation in AF. Effective prevention of obstructive respiratory events by continuous positive airway pressure ventilation (CPAP) reduces sympathovagal activation and recurrence of AF. The present review describes the relationship between OSA and AF, presents the pathophysiological mechanisms implicating OSA in AF occurrence, and provides an update of the potential therapeutic interventions for patients with OSA and AF.

journal_name

Int J Cardiol

journal_title

International journal of cardiology

authors

Goudis CA,Ketikoglou DG

doi

10.1016/j.ijcard.2016.12.120

subject

Has Abstract

pub_date

2017-03-01 00:00:00

pages

293-300

eissn

0167-5273

issn

1874-1754

pii

S0167-5273(16)34611-3

journal_volume

230

pub_type

杂志文章,评审

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