Delayed-onset hemidystonia and chorea following contralateral infarction of the posterolateral thalamus. A case report.

Abstract:

:A 68 year-old man developed progressive hemidystonia and chorea 8 months after a contralateral thalamic stroke. The neurological examination also showed a right pyramidal syndrome without hemiparesis, a right horizontal sectoranopia, and a right hemihypesthesia for all sensory modalities. The MRI revealed infarctions in the left medial temporo-occipital lobes and left posterolateral thalamus, corresponding to the vascular territories of both the thalamo-geniculate and posterolateral choroidal arterial pedicles. The thalamic lesion involved the pulvinar, the lateral geniculate body, and the ventro-postero-lateral, dorso-lateral, posterolateral, and dorso-medial nuclei, but apparently did not extent to the ventrolateral thalamic nucleus, and the subthalamic and midbrain regions. Thalamic and striatopallidal dystonia have not a common pathophysiological mechanism. The involvement of the pulvinar nucleus and of the strategic crossing of proprioceptive, cerebellar, pyramidal, and subthalamic pathways may play a role in the genesis of the posterolateral thalamic dystonia.

journal_name

Acta Neurol Belg

journal_title

Acta neurologica Belgica

authors

Gille M,Van den Bergh P,Ghariani S,Guettat L,Delbecq J,Depre A

subject

Has Abstract

pub_date

1996-12-01 00:00:00

pages

307-11

issue

4

eissn

0300-9009

issn

2240-2993

journal_volume

96

pub_type

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