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VZV multifocal vasculopathy with ischemic optic neuropathy, acute retinal necrosis and temporal artery infection in the absence of zoster rash.

Abstract：

:We describe a 54-year-old diabetic woman who developed ischemic optic neuropathy followed by acute retinal necrosis and multiple areas of focal venous beading. Vitreous fluid contained amplifiable VZV DNA but not HSV-1, CMV or toxoplasma DNA. The clinical presentation was remarkable for jaw claudication and intermittent scalp pain, prompting a temporal artery biopsy that was pathologically negative for giant cell arteritis, but notable for VZV antigen. The current case adds to the clinical spectrum of multifocal VZV vasculopathy. The development of acute VZV retinal necrosis after ischemic optic neuropathy supports the notion that vasculitis is an important additional mechanism in the development of VZV retinal injury.

journal_name

J Neurol Sci

journal_title

Journal of the neurological sciences

authors

Mathias M,Nagel MA,Khmeleva N,Boyer PJ,Choe A,Durairaj VD,Bennett JL,Mandava N,Gilden D

doi

10.1016/j.jns.2012.12.021

subject

Has Abstract

pub_date

2013-02-15 00:00:00

pages

180-2

issue

1-2

eissn

0022-510X

issn

1878-5883

pii

S0022-510X(12)00646-6

journal_volume

325

pub_type

杂志文章

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