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Experiential and genetic contributions to depressive- and anxiety-like disorders: clinical and experimental studies.

Abstract：

:Stressful events have been implicated in the precipitation of depression and anxiety. These disorders may evolve owing to one or more of an array of neuronal changes that occur in several brain regions. It seems likely that these stressor-provoked neurochemical alterations are moderated by genetic determinants, as well as by a constellation of experiential and environmental factors. Indeed, animal studies have shown that vulnerability to depressive-like behaviors involve mechanisms similar to those associated with human depression (e.g., altered serotonin, corticotropin releasing hormone and their receptors, growth factors), and that the effects of stressors are influenced by previous stressor experiences, particularly those encountered early in life. These stressor effects might reflect sensitization of neuronal functioning, phenotypic changes of processes that lead to neurochemical release or receptor sensitivity, or epigenetic processes that modify expression of specific genes associated with stressor reactivity. It is suggested that depression is a life-long disorder, which even after effective treatment, has a high rate of re-occurrence owing to sensitized processes or epigenetic factors that promote persistent alterations of gene expression.

journal_name

Neurosci Biobehav Rev

journal_title

Neuroscience and biobehavioral reviews

authors

Anisman H,Merali Z,Stead JD

doi

10.1016/j.neubiorev.2008.03.001

subject

Has Abstract

pub_date

2008-08-01 00:00:00

pages

1185-206

issue

6

eissn

0149-7634

issn

1873-7528

pii

S0149-7634(08)00033-X

journal_volume

32

pub_type

杂志文章,评审

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