Abstract:
INTRODUCTION:Chronic post-anoxic myoclonus, also known as Lance-Adams syndrome, may develop following hypoxic brain injury, and is resistant to pharmacological therapy. CASE REPORT:The patient we presented developed post-anoxic action myoclonus with severe, completely incapacitating myoclonic jerks. Myoclonus did not respond to the treatment with commonly used agents, i.e. valproate and clonazepam alone or in combination. Improvement of the action myoclonus was observed only after adding levetiracetam. CONCLUSION:Although Lance-Adams syndrome may not be fully curable at this point, levetiracetam appears to be a promising agent that can significantly improve functional level and overall quality of life of patients with this disorder.
journal_name
Vojnosanit Pregljournal_title
Vojnosanitetski pregledauthors
Božić K,Gebauer-Bukurov K,Sakalaš L,Divjak I,Ješić Asubject
Has Abstractpub_date
2014-05-01 00:00:00pages
515-9issue
5eissn
0042-8450issn
2406-0720journal_volume
71pub_type
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journal_title:Vojnosanitetski pregled
pub_type: 杂志文章
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abstract::In order to detect biochemical parameters in the peripheral blood which would serve as an indicator of severity of blast injuries, the enzyme dynamics (creatine kinase, aspartat transaminase, alanin transaminase, lactic dehydrogenase, alkaline phosphatase, angiotensin convertase) has been followed up in the serum of s...
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