Abstract:
:Chronic exposure to moniliformin results in the development of myocardial hypertrophy and degeneration. The cause of this hypertrophy is unknown. However, moniliformin-induced hypoxia or altered function of cardiac pyruvate dehydrogenase, rather than direct cardiostimulation have been proposed as potential mechanisms. Isolated guinea pig atria were used in a cumulative concentration-response model to evaluate the direct effect of moniliformin on the rate and force of atrial contraction. Moniliformin did not affect the rate or force of atrial contraction. These results are consistent with the hypothesis that moniliformin does not have a cardiostimulatory effect. Therefore cardiac stimulation. e.g. stimulation of beta-adrenergic receptors, is unlikely to be the cause of the myocardial hypertrophy observed in poultry chronically intoxicated with moniliformin.
journal_name
Mycopathologiajournal_title
Mycopathologiaauthors
Reams RY,Thacker HL,Harrington DD,Novilla MN,Wilson Bdoi
10.1007/BF00439122subject
Has Abstractpub_date
1996-01-01 00:00:00pages
115-8issue
2eissn
0301-486Xissn
1573-0832journal_volume
133pub_type
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