Abstract:
:We report a case of a cerebral arteriovenous malformation presenting as a pseudotumor cerebri syndrome. The lack of mass effect, hemorrhage, or hydrocephalus, in addition to angiographic evidence of high-flow and venous outflow restriction, implicate increased cerebral blood flow and venous hypertension as the mechanism for the papilledema. This case illustrates a pathophysiological mechanism in the development of intracranial hypertension associated with vascular malformations.
journal_name
Neurosurgeryjournal_title
Neurosurgeryauthors
David CA,Peerless SJdoi
10.1227/00006123-199503000-00020subject
Has Abstractpub_date
1995-03-01 00:00:00pages
588-90issue
3eissn
0148-396Xissn
1524-4040journal_volume
36pub_type
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